Liu, Z., Celotto, A,M., Romero, G., Wipf, P. and Palladino, M.J. Genetically encoded redox sensor identifies the role of ROS in degenerative and mitochondrial disease pathogenesis. Neurobiology of Disease, 45(1): 362-8, 2012.
Celotto, A,M., Liu, Z., VanDemark, A. P. and Palladino, M.J. A novel Drosophila SOD2 mutant demonstrates a role for mitochondrial ROS in neurodevelopment and disease. Brain and Behavior, 2(4): 423-34, 2012.
Celotto, A,M., Chiu, W.K., Van Voorhies, W. and Palladino, M.J. Modes of Metabolic Compensation during Mitochondrial Disease Using the Drosophila Model of ATP6 Dysfunction. PLoS ONE 6(10): e25823, 2011.
Hrizo, S.L. and Palladino, M.J. Hsp70 and Hsp90 mediate proteasomal degradation of TPIsugarkill that underlies pathogenesis. Neurobiology of Disease 40:676-683, 2010.
Ashmore LJ, SL Hrizo, SM Paul, W Van Voorhies, GJ Beitel and MJ Palladino. Novel mutations affecting the NA,K ATPase alpha model complex neurological diseases and implicate the sodium pump in increased longevity. Human Genetics 126: 431-447, 2009.
Celotto, A,M., Frank, A,C., McGrath, S.W., Fergestad, T.J., Van Voorhies, W.A., Buttle, K., Mannella, C.A., and Palladino, M.J. Mitochondrial encephalomyopathies in drosophila. Journal of Neuroscience 26(3): 810-820, 2006.
Palladino, M.J., JE Bower, R Kreber and Barry Ganetzky. Neural dysfunction and neurodegeneration in Drosophila Na+/K+ ATPase alpha subunit mutants. Journal of Neuroscience 23(4): 1276-1286, 2003.
Palladino, M.J., Keegan, L.P., O'Connell, M.A. and Reenan, R.A. A-to-I pre-mRNA editing in Drosophila is primarily involved in adult nervous system function and integrity. Cell 102(4): 437-449, 2000.